Skip to main content

Our Faculty

  • headshot

    Sal J. Caradonna, Ph.D., Professor and Chair

    My laboratory is interested in the post-translational mechanisms that regulate the proteins involved in base-excision repair of DNA. We are studying the aberrant pathways that lead to uracil misincorporation into DNA as well as the regulated cytosine deamination pathways of the APOBEC family of cytosine deaminases.

    Science Center, 120: 856-566-6056

  • headshot

    Katrina Cooper, Ph.D., Associate Professor

    Stress cells have to orchestrate a myriad of responses to survive or die. Incorrect choices can lead to deleterious outcomes, e.g. tumor formation. To study this, we use S. cerevisiae, human cells and mouse models. We focus on the conserved cyclin C protein that is destroyed in response to stress. Our working hypothesis is that cyclin C is a novel stress related tumor suppressor.

    Science Center, 362: 856-566-2887


  • headshot

    Renée M. Demarest, Ph.D., Assistant Professor

    The main goal of my lab is to delineate the molecular pathways required for T-cell acute lymphoblastic leukemia (T-ALL) formation, maintenance, and relapse. We are currently delineating the requirement for Notch, Ikaros, and myc in this process, and exploring novel therapeutic regimens for T-ALL treatment. In addition, we are developing novel methods to treat various murine models of cancer with human equivalent chemotherapeutic regimens in order to increase the translational success of new therapeutics to the clinic.

    Science Center, 276: 856-566-6402

  • headshot

    Ronald Ellis, Ph.D., Professor

    Control of Germ Cell Fate: Animals must produce sperm or eggs to reproduce. Although these cell types differ dramatically, they are produced from similar progenitors. Understanding how this process is controlled could revolutionize our ability to treat reproductive disorders and infertility in humans. Evolution of Hermaphroditism: Sexual traits are among the most rapidly changing features of each species. To learn how these changes take place, and how developmental pathways constrain which ones occur, we are studying the evolution of mating systems in nematodes.

    Science Center, 316: 856-566-2768


  • headshot

    Jennifer Fischer, Ph.D., Vice Chair for Education

    We are evaluating the role and value of biomedical knowledge in clinical reasoning and diagnostics by obtaining feedback from practicing physicians. We also focus on understanding and utilizing educational technologies such as exam software, online learning management systems, and virtual programs to enhance student learning.

    Science Center, 390A: 856-566-6919

  • headshot

    Gary S. Goldberg, Ph.D., Associate Professor

    Cells must communicate with each other to coordinate the development and survival of an animal. This communication can be mediated by diffusible factors that pass between cells, or by direct contact through cell junctions. I am interested in how intercellular communication affects cell growth and differentiation, with an emphasis on how cell communication can control tumor cell growth and prevent eye diseases.

    Science Center, 320: 856-566-6718

  • headshot

    Michael Henry, Ph.D., Assistant Professor

    We use the yeast Saccharomyces cerevisiae as a model system to understand the molecular mechanisms by which RNA precursors are processed in the nucleus. More precisely, our goal is to understand the role of posttranslational protein modification in this process.

    Science Center, 320: 856-566-6970

  • headshot

    Ping Jiang, M.D., Assistant Professor

    My laboratory, the ES Cell and Transgenics Laboratory at Rowan University School of Osteopathic Medicine, creates genetically modified mouse models for studies of human diseases and is open to the scientific communities in the United States as well as around the globe. Numerous transgenic, knockout and knockin mouse models were generated when I directed similar facilities at The Wistar Institute and Cold Spring Harbor Laboratory for over ten years.

    Director, ES Cell and Transgenics Laboratory: 856-566-7330


  • headshot

    Kai Mon Lee, Ph.D., Associate Professor

    We use 3D printing to fabricate three-dimensional (3D) scaffolds of varying architecture to provide 3D organization of neurons and their supporting cells with the objective of mimicking their 3D environment in the human brain. The scaffolds are constructed of biocompatible protein and protein composites. We use these platforms to study neurodegenerative diseases such as Alzheimer’s and Parkinson’s and, drug delivery. Another research interest of our lab is the fabrication of implantable protein-based neuron chips.

    Rowan Medicine Building, 2214: 856 566-6152

  • headshot

    Katharine Milani, Ph.D., Assistant Professor

    Dr. Milani received her doctoral degree in Cell and Molecular Biology from Thomas Jefferson University where she studied the human malaria parasite, Plasmodium falciparum. At her previous institution, where she served as department chair, she coordinated the biotechnology program and researched disease burden in local mosquito populations; additionally she oversaw the implementation of an online anatomy course with lecture and lab components. Her current focus is on innovative instructional delivery and assessment design, specifically in the areas of microbiology and histology.

    Science Center: 856 566-6041

  • headshot

    Eric Moss, Ph.D., Associate Professor

    We study developmental timing, microRNAs and translational control in C. elegans and the mouse. The worm heterochronic gene lin-28 is regulated by microRNAs and encodes a specific mRNA-binding protein. Its human homologue, Lin28, appears also to be a microRNA-controlled developmental regulator.

    Science Center, 312: 856 566-2896

  • headshot

    Susan Muller-Weeks, Ph.D., Assistant Professor

    Research in the laboratory focuses on the repair of uracil in DNA, which is critical for the maintenance of genomic integrity. Specifically we are elucidating transcriptional and post-translational pathways that regulate expression of uracil-DNA glycosylase under normal cellular conditions and in response to anti-tumor agents.

    Science Center, 130: 856 566-6097

  • headshot

    Catherine L. Neary, Ph.D., Assistant Professor

    Altered organelle function initiates a cell stress response; failure of this stress response results in the coordinated disassembly of the cell, known as apoptotic cell death. My interest is how metabolic stress affects the initiation of cell death, and how organelles, specifically the mitochondria and cytoskeleton, communicate during the death process. Additional projects in my laboratory include assessing the role of lactic acid metabolism in mitochondrial function, as well as cancer cell dependence on abnormal AMP-dependent kinase (AMPK) activation.

    Science Center, 313/314: 856 566-6373

  • headshot

    Randy Strich, Ph.D., Professor

    Our laboratory focuses on understanding how the transcription program is coupled to meiotic progression in budding yeast. A second project investigates the activity of the conserved C-type cyclin in directing the oxidative stress response and apoptosis in yeast and mammalian systems.

    Science Center, 354: 856-566-6043

  • headshot

    Brian P. Weiser, Ph.D., Assistant Professor

    My lab examines the architecture and function of protein-ligand and protein-protein complexes that are mediated by low-affinity interactions. We use chemical and molecular techniques to quantify and manipulate low-affinity interactions to examine their biochemical relevance. Current projects examine (1) DNA repair protein complexes and (2) neuronal deacylase enzymes.  

    Science Center, 307A: 856-566-6270